SpiritualONE
Head Administrator
Registered: 02-2004
Posts: 25404
Karma: 4 (+4/-0)
|
|
Reply | Quote
|
|
Re: CoQ10 Helps With Many Health Problems
diabetes Mellitus:
definition: A disease of carbohydrate metabolism due to a lack, or insufficient action, of circulating insulin thus causing hyperglycemia and known sequelae." etiology: Diabetes mellitus (DM) is a leading cause of death in the U.S. and its incidence is rising. DM has been linked to the Western diet and lifestyle (particularly high carbohydrate intake).
Diabetes word for today:
sequela[sikwē′lə] pl. sequelae
Etymology: L, sequi, to followany abnormal condition that follows and is the result of a disease, treatment, or injury, such as paralysis after poliomyelitis, deafness after treatment with an ototoxic drug, or scar formation after a laceration.
Other theoretical causes for the deficiency of insulin include viral infection of the beta cells, toxic reaction to N-nitroso compounds (found in smoked and cured meats), and auto-immune reaction.
DM is classically split into two types, insulin-dependent, called Type I, and non-insulin-dependent, called Type II. The two types vary markedly in etiology, pathophysiology, course and complications. Other types are also detailed:
• insulin dependent DM (Type I): These patients are susceptible to ketosis and have certain HLA antigens as well as pancreatic islet cell antibodies. Typically, this type of DM affects young people with peak occurrences at 5 years old and then again between 10 and 12 years old. It accounts for only about 10% of the diabetic patients in the U.S. Presentation is usually with ketoacidosis and history of recent weight loss. Genetic susceptibility and a viral infection prompting immune targeting of pancreatic beta cells is a suggested mechanism for development of Type I DM. Recent research indicates that cow’s milk proteins may be an inciting antigen to trigger off autoantibodies to pancreatic cells. (Maclaren N, Atkinson M. NEJM vol 327:5, July 30, 1992.)
• non-insulin dependent DM (NIDDM): These diabetic patients are not susceptible to ketosis. Most are obese and between 50-70 years old at onset. While the mechanism in Type I is the lack of insulin, in Type II the cause seems to be cellular resistance to available insulin.
• DM from certain conditions: for example, pancreatic disease, malnutrition, etc.
• gestational DM: glucose intolerance during pregnancy, whether present but unknown before pregnancy or developed post-conception.
• impaired glucose tolerance (IGT): Patients who have blood results in-between the normal and diabetic reference ranges.
I normally don't do this but I'm going to put down this treatment plan that has some controversial foods. Tell me if you can pick them out? » eating principles • Decrease sucrose and other simple carbohydrates: all simple sugars, including fructose, should be markedly lowered. Fructose is now being touted as a "good" food for diabetics because of its lack of insulin stimulation. However, a number of studies have shown that fructose consumption may also result in long term deleterious effects.
Note: In acute loading studies a synergism between glucose and fructose with regard to insulin secretion exists. Neither glucose or fructose, when given as the sole monosaccharide, stimulates insulin secretion as potently as glucose and fructose combined. (Beck-Neilsin, et al. AJCN. 33:273; Hallfrisch, J. et al. AJCN 43:151-59, 1986.) • Increase complex carbohydrates to 60-70% of caloric intake; decrease fat to 20% (Chait, A. Contemp. Nutr. (2), Feb,1984) • Increase consumption of foods with low glycemic indexes (especially legumes). Legumes in the morning have shown an extremely beneficial effect on blood glucose; in fact, they seem to keep the blood glucose at a fairly low level for almost 4-6 hours. • Treat food sensitivities • Fiber — foods high in water-soluble fiber: up to 80-100 gms/day. guar gum (5g/meal) pectin - up to 30gms/day oat bran (1 cup oats provides 9gms fiber) » for juvenile onset: • avoid dairy: cow's milk proteins may be an inciting antigen to trigger off autoantibodies to pancreatic cells. (Maclaren N, Atkinson M. N Engl J Med 327(5):348-349.) » for adult onset : • low sugar, low cholesterol • low fat diet of unsaturated fats • calorie percentages: 70% complex carbohydrates, protein 12-15%, fat 15-18% • high fiber • low Sodium/Sodium-restricted diet • avoid overeating! If overweight, reduce weight to normal range. Avoid mental and nervous stresses, especially worrying. • eat in a pleasant environment and chew all food well. Eat 5-6 small meals through out the day instead of large meals. • alkaline vegetarian diets can be helpful • elimination/rotation diet, rotation diet, rotation diet expanded therapeutic foods: • cucumber, stringbeans (Airola, p. 72) • garlic, Jerusalem artichokes, burdock, parsley (Shefi) • foods rich in Chromium and Zinc (Airola, p. 72) • foods high in water-soluble fiber: flax seed, pectin, guar gum, oat bran, mucilage • complex whole grain and legume carbohydrates • pumpkin, whole rice, yams, mung beans, squash, celery, peach, millet, onion, spinach, blueberry, peas, tofu, cabbage, daikon radish, mulberries (Ni, p. 122) • foods rich in Iodine, Silicon, Phosphorus: kelp, dulse, Swiss Chard, turnip greens, egg yolks, wheat germ, cod roe, lecithin, sesame seed butter, seed and nuts, raw goat milk (Jensen, p. 61) • garlic, wheat germ, liquid chlorophyll, alfalfa sprouts, buckwheat, watercress, rice polishings, apple, celery, cherries (Ni, p. 120) • onions, beans, legumes, soy, ginger, alfalfa, yogurt, brewer's yeast (Marz) • increase omega-3 and omega-6 fatty acids: vegetable, nut, seed oils, salmon, herring, mackerel, sardines, walnuts, flax seed oil, evening primrose oil, black currant oil • spices: cinnamon, turmeric, bay leaf, cloves » Kidney Xu (Deficiency) Diabetes: • foods that tonify the Kidney » Spleen and Stomach Xu (Deficiency) Diabetes: • harmonize Spleen/Stomach foods fresh juices: • stringbean, cucumber, and celery (Airola, p. 71) • raw sauerkraut and lemon • raw sauerkraut and tomato • parsley tea • huckleberry leaf tea (Shefi) • stringbean, brussel sprout, carrot, and lettuce (Walker, p. 77) • watermelon and tomato (Chao-liang, Qing-rong, Bao-zhen, p. 62) eating principles for Juvenile-Onset, Insulin-dependent : • diet should be carefully calculated according to the type and amount of insulin used. Children need enough energy for growth, so supply adequate calories from complex carbohydrates. All other information concerning Adult-onset is applicable. fresh juices: • bamboo shoots and celery, warm and drink (Ni, p. 33) celery (Ni, p. 33) specific remedies: • celery, yam, and pumpkin (Ni, p. 33) • pumpkin, yam, and potato pie (Ni, p. 122) • cook snow peas, blend into juice, drink 1/2 cup, twice daily (Ni, p. 47) • tea from spinach and chicken gizzards; l cup, three times daily (Ni, p. 49) • soup from cabbage, yam, winter melon, and lentils (Ni, p. 122) • steam tofu, cool, add sesame oil and slices of raw squash (Ni, p. 122) • soup from mung beans, peas, and barley (Ni, p. 122) • steam millet with yams and a few dates (Ni, p 122) • drink daikon radish, celery, carrot, and spinach juice (Ni, p. 122) • crack open a fresh coconut and drink the juice, twice daily (Yin-fang and Cheng-jun, p. 86) See Exchange lists for diabetes avoid: • food intolerances. (Pots J., Lange M. Diabetes, vol26, supp. 1 #234, 1977 and Diabetes 29, 6, 1980; Philpott and Kalita, 1983) • coffee (Tuomilehto J., et al. Brit Med Journal 300:642-3, March 1990.) • sugars (all types: malt, maltose, dextrose, corn syrup), candy, honey, molasses, dried fruits, concentrated sweets, concentrated juices, fried foods, caffeine, spicy foods, processed foods • trans-fatty acids, hydrogenated oils (margarine, vegetable shortenings, imitation butter spreads, most commercial peanut butters) oxidized fats (deep fried foods, fast food, ghee, barbequed meats) • Iron: Excess iron may stimulate the production of free radicals which can damage the beta cells of the pancreas, damage the insulin molecules that are secreted, or in some way effect the insulin receptors. (Phelps, Chapman, Hall, Brand, and Mackinnon. Lancet 2:233-34, 1989; Cutler, P. Diabetes 38:1207-10, 1989.)
|
